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For several years now it has been a criticism by those who are opposed to the Brewer diet and philosophy that his writings
are outdated because they speak so frequently of the hazards of diuretics. These critics maintain that there are no mainstream
doctors who use diuretics any more, and that therefore the Brewer people are out of touch with the current needs of pregnant
women. My answer to these critics has been that although few if any doctors use diuretics on normal pregnant women these
days, Brewer's basic premise is still valid, and that today's doctors still use other means to try to control the weight gain,
blood pressures, and edema of their pregnant women--other means such as low-salt, low-calorie, low-protein diets, and sometimes
anti-hypertensive medications, which are just as hazardous to pregnant women as the diuretic therapies were.
However, it has come to my attention recently that there seems to be a new threat that has grown in our country, from a new
source of diuretics which I was not fully aware of until now. It has come to my attention that there seems to be a large
number of pregnant women who are using herbs which have diuretic properties during the course of their pregnancies. The herbs
which I have become aware of so far are nettle, dandelion, alfalfa, bilberry, and celery. Some of these herbs are found in
pregnancy teas, some in liver cleansers, some in iron preparations, and some are used for other reasons. Some women seem
to be actually intentionally using these herbs for their diuretic properties--in an attempt to lessen the swelling (edema)
that they may be experiencing in their ankles, or in an attempt to lower their rising blood pressures.
There are some who would argue that herbal diuretics are less harmful and harsh than prescription diuretics are. There are
some who would argue that these herbs with diuretic properties also provide vitamins which are essential to the good health
of the liver and kidneys and various functions of the pregnant body. There are even some who would argue that a little bit
of a diuretic action can't possibly harm the pregnant woman.
I think that it is quite possible that at some level, herbal diuretics may be less harsh to the body generally than prescription
diuretics are. I also think that it is quite likely that these herbs are high in vitamins which are very beneficial to the
pregnant body, and which could be very healthy for the liver or kidneys, or could be a good iron source for the blood.
What I am very dismayed to discover, however, is the strong assertion by some herbalists that surely a little bit of diuretic
action by some herbs can't possibly harm a pregnant woman, especially those who might be a little uncomfortable due to some
swelling in her ankles or legs, or those with blood pressures that are creeping up. It is clear to me that those who believe
this assertion don't understand what the pregnant body is trying to do in order to remain healthy.
It is vitally important for everyone who cares for pregnant women, or who creates supplements or teas for pregnant women,
or who sells any form of herbs to pregnant women, to understand that one of the most important functions of the pregnant body
is to increase the mother's blood volume by 40-60% over the course of her pregnancy--and more than that if there is more than
one baby. That extra blood volume is vital for the healthy implantation and function of the placenta, and for the healthy
function of the liver and kidneys, and for the adequate nutrition of the baby, the placenta, and the uterus.
If the blood volume is too low for pregnancy, when it does not increase by 40-60%, due to inadequate nutrition, or due to
the use of diuretics (herbal or prescription), the kidneys secrete a substance called renin. Renin is a substance that the
kidneys secrete at any time that the blood volume is below normal, whether a person is pregnant or not. The action
of renin on the body is to constrict the capillaries, for the purpose of sending most of the inadequate blood supply to the
vital internal organs, to preserve the life of the body. In pregnancy, this renin response by the kidneys to a lower-than-normal
blood volume causes the mother's blood pressure to rise. Making the blood volume drop even more by giving the mother diuretics
(either herbal or prescription), makes her kidneys secrete more renin, which makes her blood pressure rise even higher.
To say that in another way--diuretics in any form force the kidneys to lower the mother's blood volume by removing more
fluid from her blood and losing it in the form of urine. A lower blood volume triggers the secretion of renin, which causes
a rise in blood pressure. Thus, in normal pregnancy the use of diuretics from any source causes a rise in blood pressure
and the beginnings of the pre-eclampsia process.
Pathological swelling (edema) in pregnancy is also caused by an inadequate blood volume, and it is also made worse by the
use of diuretics, regardless of the source of those diuretics. Much of the swelling/edema in pregnancy is normal, or physiological--caused
by the hormones of pregnancy and the weight of the baby limiting the return of the blood flow from the legs to the heart.
But when the mother is on an inadequate diet, or on diuretics, the loss of fluids from the blood can also cause pathological
swelling/edema.
This pathological edema is caused by another response by the kidneys which is triggered when the blood volume is too low.
This second response is an effort by the kidneys to conserve fluid by sending less fluid to be expelled with the urine.
The kidneys send this conserved fluid back to the blood stream, in an attempt to increase the blood volume to more normal
levels. If there is not enough osmotic pressure in the blood to hold this conserved fluid in the blood stream, osmotic pressure
normally created by the presence of albumin and salt in the blood, this conserved fluid will not stay in the blood stream.
Instead, it will move out of the capillaries into the tissues in the ankles, legs, fingers, and face. This is what causes
the pathological swelling/edema in pregnancy. The use of diuretics to try to force the fluid out of the tissues, and to force
the kidneys to lose this fluid in the urine, only makes the blood volume fall even more, which eventually causes even more
swelling/edema as the kidneys try to compensate by conserving more fluid.
It is vitally important for pregnant women to understand, and for those who care for them and supply them with herbs and supplements
to understand, that there is a huge difference between the edema and hypertension of non-pregnant people with heart disease,
kidney disease, and circulatory disease, and the edema and hypertension of normal, otherwise-healthy pregnant women. The
edema and hypertension of the diseased body is caused by an abnormally expanded blood volume, and that condition
must be treated with various therapies which help the body deal with that expanded blood volume--therapies that may include
diuretics. The normal pregnant body that is developing pathological edema or hypertension is suffering from an abnormally
contracted blood volume, and the only way to turn that condition around is to assist the body in its efforts to expand
that blood volume. Using diuretics counteracts the pregnant body's efforts to increase the blood volume. Helping the pregnant
mother to eat more calories, more salt, and more protein is the therapy which will help her body expand its blood volume to
the level that is needed for sustaining a healthy pregnancy.
One of the great tragedies of this situation is that many of the mothers using and seeing the effects of these diuretic herbs
may have been working very hard to follow the Brewer Diet--a nutritional plan that they expected to keep their blood volume
expanded, a nutritional plan that they expected to help them prevent the PIH, pathological edema, pre-eclampsia, IUGR, premature
labor, abruption of the placenta, and low birth weight babies that result from an abnormally contracted blood volume in pregnancy.
They may have been also taking these herbs to help feed and sustain their livers, which is actually another goal of the Brewer
diet and philosophy. Little did they know that by taking herbal diuretics they were actually undoing much of their diligent
nutritional work with which they'd intended to keep their blood volume expanded and healthy.
So I appeal to the herbalist community to take up the challenge of warning all pregnant women to take care to not use any
herbs that have diuretic properties. I urge them to remove herbs such as nettles, dandelion, alfalfa, bilberry, and celery
from their pregnancy teas and their pregnancy supplements. I plead with them to find other non-diuretic herbs with equally
nutritious qualities, for their iron supplements. I appeal to them to post warnings on their websites, alerting pregnant
women to the hazards of using various liver-cleanse or kidney-nurturing supplements if they contain dandelion, nettle, alfalfa,
bilberry, or celery, or any other herb with diuretic properties. Let us all work together and assist each woman to have as
healthy a pregnancy as possible, by helping each woman to grow as healthy a blood volume as possible.
(Joy Jones, April 9, 2008)
See here for information about some of the common misconceptions regarding the Brewer Diet
See here for information about how the Brewer Diet can prevent complications in pregnancy
The following is reprinted from What Every Pregnant Woman Should Know, by Gail Sforza Brewer with Tom Brewer,
M.D., 1977.
"Understanding Swelling: water retention is normal" (p. 34)
Eighty to ninety percent of women swell up at some time in the course of their pregnancies. Most American obstetricians
look on this normal swelling with alarm. The spectre of toxemia is never far from their minds, and toxemic women swell up.
Physicians have been trained to view swelling as a potential danger sign. When they see swelling of the face or hands, they
recoil in horror. This is definitely a "condition" to be "treated." They attack the swelling with therapeutic frenzy. They
de-salt. They drug. They dehydrate. Then they are confounded when their patients develop toxemia, anyway.
Dr. Leon Chesley, distinguished author of the toxemia chapter in Williams Obstetrics, the most widely used obstetrics
textbook, now challenges this traditional approach to pregnancy swelling. After forty years of research in the field, he
has concluded that normal swelling, or physiologic edema, is a sign of health in pregnant women, and not a pathological condition.
At a July 17, 1975, hearing of the Food and Drug Administration on the use of "water pills," or diuretics, in pregnancy, Dr.
Chesley testified that 60 to 70 percent of normal pregnant women will have benign swelling of their faces and hands--in addition
to that of their feet and ankles.(1)
This single statement is of enormous significance because up to two million pregnant women a year since 1958 [as of 1977]
have been placed on potent diuretics to "treat" the very edema Professor Chesley termed normal.
Citing study after study, going back as far as Dexter and Weiss's classic book on toxemia (1941), Dr. Chesley criticized the
routine American obstetrical practice of "treating" pregnancy edema at all. Instead, he argued for an appreciation of its
underlying physiologic causes.
Normal water retention comes about in pregnancy chiefly from an impressive rise in the level of female hormones, principally
estrogens, manufactured by the placenta. These hormones are the same ones which cause many women to have water build-up and
swelling in the few days preceding their menstrual periods, or when they are taking birth control pills. During pregnancy
these hormones influence connective tissue throughout the body to retain extra fluid. Hence, the pregnant women commonly
experiences swelling of her face and hands (generalized edema) in addition to that of her feet and lower legs (dependent edema).
The retained fluid is of benefit to mother and baby. Like a reservoir, it provides a water storage system in the mother's
body. The stored fluid serves as a safeguard, a backup for the expanded blood volume we have learned is needed to nourish
the placenta. At the time of the birth, when some blood loss is unavoidable, the extra fluid protects the mother from going
into shock. Remaining tissue fluid is mobilized in the early breast-feeding period to insure the mother an adequate milk
flow.
In women pregnant with twins, the process of physiologic swelling is exaggerated. Their larger placentas manufacture more
hormones, which cause more water to be retained in their bodies--normally! This additional water, plus the weight of the
second baby, dramatically increases the weight gain of the mother carrying twins. Weight gains of fifty to sixty pounds are
typical when mothers are encouraged to eat well. Unfortunately, in the United States, where rigid weight control, salt restriction
and diuretic therapy have characterized standard prenatal care, diagnosis of a twin pregnancy automatically assigns a mother
to the so-called "high-risk" category. It is easy to understand why twins have had so much trouble when their intrauterine
growth has been consistently subverted by these practices. It has even come to be accepted by doctors and mothers alike that
"twins come early"--that they are born three or four weeks ahead of time, and that each must weight less at birth than a single
infant would. People have the idea that the mother's uterus had stretched as much as it could--"there was no more room"--so
the babies had to be born.
When mothers of twins are counseled to eat correctly for three throughout gestation they meet their increased nutritional
demands. When they refuse diuretics and low-salt diets for their extra physiologic edema they usually give birth, at term,
to infants of normal birth weight. Twins are not of necessity "high-risk." They only become so when management incompatible
with physiology is imposed by the physician.
Dr. Chesley, in his FDA testimony, consistently associated the presence of physiologic edema with better infant outcome.
On two critical measures, birth weight and infant mortality, mothers with normal swelling did far better than those without
it.
Drawing attention to a major conclusion of the 1968 NIH Collaborative Study of Cerebral Palsy, Dr. Chesley noted that
babies born to mothers with normal swelling were of higher birth weight than those born to mothers with no swelling.
The Collaborative Study also found that a baby's birth weight is the most reliable indicator of future neurologic development.
Low-birth-weight babies have a much higher likelihood of starting life with significant brain damage or growing up to face
learning difficulties in school.
Dr. Chesley also reported a review of the medical records of 17,000 American mothers pregnant for the first time. In this
study edema was associated not only with higher birth weight, but also with lower infant mortality. In 10,126 mothers who
at no time had edema of the hands or face, the infant death rate was 26 per thousand. In the 6,963 mothers who did have edema
of hands and/or face, the infant death rate was 18 per thousand. There was 44 percent higher infant mortality in the no-edema
group.
After presenting this evidence and a very erudite discussion of the other harmful effects of "water pills" (which called into
question the validity of the research which had originally persuaded the FDA to allow them to be used in pregnant women),
Dr. Chesley went on record in opposition to the use of diuretics in human pregnancy. He stipulated only one exception to
the blanket contra-indication. Diuretics may appropriately be used when the mother suffers heart failure, kidney malfunction,
or other medical disease which results in abnormal water retention in both the tissues and the circulation.
This exception does not apply to toxemia, Dr. Chesley asserted. He adamantly stated that diuretics do not prevent or ameliorate
toxemia. This bold conclusion descredited the slick, four-color spreads promoting diuretics which have appeared in every
American OB/GYN journal since 1958. To the contrary, Dr. Chesley blamed diuretics for aggravating a significant abnormality
present in mothers with toxemia, low blood volume (hypovolemia). The diuretics act to drive salt and water from the circulation,
thus shrinking the blood volume even more. When used in conjunction with a low-salt diet from early pregnancy on, as the
drug companies urged in their promotions, the diuretics may actually bring on the toxemia the doctor seeks to prevent.
What has been the outcome of this hearing? Up to now, most practicing obstetricians do not even know it was held. No testimony
from the several physicians who appeared at the hearing has been publicized. The FDA has not called a public press conference
to warn our public directly about the hazards of these drugs, even though millions of women and unborn babies continue to
be exposed to them. Nor have the customary warnings been sent to physicians as was done recently after the disclosures that
certain hormones often used to prevent spontaneous abortions cause vaginal cancer in female children born to mothers who took
them in early pregnancy. Rather, the FDA has merely issued regulations requiring a change of labeling on the drugs, removing
the indication that they are effective in toxemia. Most obstetricians practicing today have been trained to use these drugs
as part of routine pregnancy management. Without special warnings, this labeling change in the fine print of the doctors'
portion of the package insert will probably go unnoticed by the busy physician. Alarmingly, the American College of Obstetricians
and Gynecologists, whose representative at the hearing argued that the drugs should continue to be prescribed if the mother
is "too uncomfortable" at the end of pregnancy due to edema, still clings to this position [as of 1977]. As a result, many
thousands of women each year will continue to take these drugs because their doctors will continue to write the prescriptions.
Without the correct information from their physicians about normal swelling, many women are dismayed by the way they look
when they begin to swell a bit. Many physicians play on the mother's glum assessment of her looks as a way of forcing compliance
with their low-salt diets and diuretics. If the mother refuses to cooperate, other forms of pressure may ensue. She is often
told that her swelling is related to unnecessary accumulation of fat during pregnancy which will lead to permanent obesity.
Or that her husband might lose interest in her if she becomes obese. The mother, not realizing that her swelling is probably
normal and will vanish after the baby is born, accepts her doctor's appraisal.
One suburban mother angrily recalls how her obstetrician was so disgusted with her twenty-eight-pound weight gain and open
disregard for his diet during her second pregnancy that he refused, point blank, to attend her delivery. He "taught her a
lesson" by leaving her in the hands of an inexperienced resident she had never met before!
Her healthy baby boy weighed seven pounds--a marked difference from her first child, who weighed three and a quarter pounds
and was born prematurely after an induced labor due to toxemia. This mother had followed the doctor's diet the first time,
and the child has had an endless series of health problems since birth, a victim of intrauterine malnutrition.
Popular women's magazines stacked in the doctor's waiting room are of no help, either. Their pages are full of advertisements
for mild diuretics to relieve swelling before a woman's period, or for "quick weight loss" when her favorite dress is a little
too tight. Diet soda and junk food layouts promise satisfaction without nutrition. A barrage of underweight models promote
emaciation as the American standard of beauty. Each issue rhapsodizes over the latest Hollywood diet guaranteed to keep readers
vibrant and sexy while subsisting on only grapefruit, only rice, or only fluids. Little wonder the pregnant woman is on the
defensive about her size and shape for nine straight months! No wonder she worries about swelling.
When swelling becomes uncomfortable, as it might toward the end of the pregnancy, the mother should take the following steps:
- Switch to open, flat shoes like summer sandals. Feet are then free to swell as the day goes on, not pinched tight in
closed shoes.
- Try to minimize chair-sitting, especially on hard surfaces. Return of blood from the lower legs is impeded as the chair
edge presses into upper leg. Sitting tailor-style (cross-legged) or using an ottoman for a footrest brings lower legs even
with hips, assisting the flow of blood.
- Lie with feet elevated on pillows, permitting return of blood pooled in feet and lower legs. Repeat three or four times
a day, five to ten minutes each time.
- Keep salting food to taste. Swelling can result from too little salt in the diet.
If the doctor suggests diuretics at any time in pregnancy, the mother must ask questions.
First, of herself: Am I eating a good, balanced diet for pregnancy? Am I getting enough protein, calories and salt? Swelling
can result from deficiencies of any of these nutrients.
Next, of the doctor: Do I have any medical disease which causes an abnormal increase in blood volume, such as heart failure
or nephritis? Diseases in which excess fluid is retained in the circulation may be aided by judicious diuretic therapy.
An internist should be consulted and careful evaluation of the mother's condition made if any of these medical diseases are
suspected. The good obstetrician recognizes his limitations and will seek consultation from other specialists when indicated.
Women must know that these diseases are exceedingly rare during the childbearing years. So rare, in fact, that if a doctor
prescribes a diuretic for her, she must ask why she needs it. If he assures her she has no abnormal increase in her blood
volume due to underlying medical disease, she should refuse to take the pills. Diuretics can do nothing but harm except in
these rare situations.
Dr. Douglas R. Shanklin, professor in both the departments of OB/GYN and Pathology at the University of Chicago Medical School
and past editor of the Journal of Reproductive Medicine, declared in 1973:
Modern renal physiology makes it clear that the use of diuretics in pregnancy has little or no basis. There is a strong
body of belief that they are causative of complications. The use of diuretics in pregnancy should be banned; they should
be abandoned in modern prenatal care.
What Every Pregnant Woman Should Know available here
The following is reprinted from Metabolic Toxemia of Late Pregnancy: A Disease of Malnutrition, by Thomas
H. Brewer, M.D., 1966 & 1982.
"Dietary Salt and Diuretics" (p. 72)
My own clinical experiences working with many normal and toxemic pregnant women have led me to the firm conviction that restriction
of salt in the diet of pregnant women produces no clinical benefit. Several investigators in this country and in England
and in Canada have recorded similar experiences. 43,44,45 Of course this does not apply to the women with significant cardiovascular
or renal disease during pregnancy.
Salt restriction has some undesirable results, particularly when combined with the use of saluretic diuretics. Many women
have told me that both physicians and public health nurses had told them not to drink milk because it contains too much salt.
This is wrong, because milk is one of the most important and cheapest sources we have available for high biological quality
proteins. A low salt diet is not very savory, and the patients often do not eat well when actually following such a diet.
It is in the hospitalized patients that one of the most glaring errors is often made in pregnancy nutrition. Here we have
opportunity to provide the patient with an optimum diet planned and prepared by expert nutritionists. I have been in several
hospitals in our nation where the routine management of the toxemic patient calls for a "low salt diet" which on inquiry is
found to contain only 50 gm of protein. To reduce the toxemic patient's protein intake below that of the requirements of
normal pregnancy is to make a grave physiological and biochemical mistake.
Figure 11 (Chap. 4, p. 52) demonstrates a common clinical phenomenon: a diuretic which causes the kidneys to excrete an excessive
amount of sodium and potassium, and water associated therewith does not have any effect upon the underlying metabolic disorder
in MTLP, for as soon as the diuretic is stopped, the sodium and water retention immediately recurs. A diuresis may blind
the physician to the fact that the patient is really getting worse. Diuretics are absolutely contraindicated in the severely
toxemic patient who has a contracted blood volume, low serum albumin and hemoconcetration. The following three cases [to
be added to this website at a later date] are presented in detail to illustrate the clinical reality of these ideas. It was
from the careful study of these and other similar cases that I began to crystallize my ideas about the pathogenesis of metabolic
toxemia of late pregnancy and to turn from concentration upon sodium, water, diuretics and the kidneys to concentration upon
nutrition and hepatic dysfunction.
43. Robinson, Margaret: Salt in pregnancy. Lancet, 1:178 (Jan. 25), 1958.
44. Mengert, W.F., and Tacchi, D.A.: Pregnancy toxemia and sodium chloride, Amer. J. Obstet. Gynec., 81:601, 1961.
45. Bower, David: The influence of dietary salt intake on pre-eclampsia. J. Obstet. Gynec. Brit. Comm., 71:123, 1964.
Metabolic Toxemia of Late Pregnancy available here
The following is reprinted from Medikon International no. 4 - 30-5-1974.
"Iatrogenic Starvation in Human Pregnancy", by Tom Brewer, M.D., County Physician, Contra Costa County Medical Services,
Richmond, California, U.S.A.
Frank Hytten tells us in his pregnancy physiology textbook of two pioneers in the field of iatrogenic starvation in human
pregnancy.(1) A certain Professor Brunninghausen of Wurzburg decided, for reasons unstated, that it was better for women
to eat less food during pregnancy; this was in 1803. A century later Prochownick is given credit in 1899 for introducing
the idea that caloric and fluid restrictions during human pregnancy could produce an infant who weighed less at birth.(2)
The intention of Prochownick was to minimize the cephalopelvic disproportion in a woman with a borderline contracted pelvis
and thus reduce the incidence of surgical intervention in such patients. As surgical techniques developed with the practices
of asepsis and improved anesthesia established, Western European obstetricians lost all fear of operative delivery. Prochownick's
valuable clinical observation that caloric and fluid restrictions do in fact lower the birth weight of the newborn human infant
was forgotten. Thus a very important clue to the mystery of "low birth weight for dates" newborns was buried.
Unfortunately, this still universal misconception became established as a dogma in clinical obstetrical teachings in Western
medical culture: the human fetus is a parasite, will grow according to its "genetic code" to a given weight and length before
birth, and that this growth and development are in no concrete, material sense influenced by the foods and fluids the pregnant
woman is taking in during the course of her gestation.(3,4) Scientific obstetrics still suffers today from what I term "nutritional
nonchalance" related to this false belief.
In 1972 officially in the United States the cause of eclampsia, the disease I term convulsive metabolic toxemia of late pregnancy
(MTLP), was "unknown."(5,6) [This official assertion continues into 2008] Since this dread disease remains a common cause
of maternal, fetal and newborn morbidity and mortality throughout the world, speculations about its etiology continue. It
has been long believed that the Nutrition of the pregnant woman during gestation does in fact influence her development
of MTLP. Women who develop MTLP are still accused of eating too many calories and too much salt (NaCl). That such
an idea remains popular in 1973 stems from the fact taht very few Western-trained OB/GYN surgeons have ever taken time or
interest to ask these poor women what foods and how much they have been eating and drinking during pregnancy.
When I began to work in the Tulane Service's prenatal clinics at Charity Hospital, New Orleans, Louisiana, as a third year
medical student, pregnant patients were being told to restrict their caloric intake and to restrict their dietary salt intake:
"So you won't have fits....so you and your baby won't die from toxemia." It is difficult for me to learn what is happening
there now since no members of the Tulane faculty will communicate with me, but unofficial sources informed me not long ago
that "...nothing has changed in this field since you were here over 20 years ago." I studied this problem for four years
in another city-county hospital in our deep south. Jackson Memorial Hospital, Miami, Florida, from 1958 to 1962.
A reliable communication from an established ostetrician in Miami in March, 1973 informed me that "...nothing has changed
in this field since you left here over ten years ago." The common practices of weight control and dietary salt restriction
seem eternal.
Since it is now clear that the sudden, rapid weight gain observed in patients with severe MTLP is a result of malnutrition
with a falling serum albumin concentration, hemoconcentration, a falling blood volume with increasing interstitial fluid,
we no longer need to blindly "control weight" with starvation type diets. However, fear of the unknown drives the most rational
and "scientific" people to irrational actions; millions of pregnant women in Western European medical culture still suffer
from iatrogenic starvation diets in the vague hope that caloric and salt restriction will in some way protect them and their
unborn from the "ancient enigma of obstetrics," eclampsia.
Iatrogenic starvation in human pregnancy has a long and ignoble history in the United States: its traditions run strong and
deep in one of our oldest and most respectable journals of obstetrics, the American Journal of Obstetrics and Gynecology.
In its second volume published in 1921 we find this account by Rucker:
"On August 2nd, two weeks after her first visit, her blood pressure was 120/80, the urine was free from albumin and sugar.
On August 17 her weight had increased 6 pounds and her legs were swollen up to her knees. She had no headache. Blood pressure
was 180/90. Urine was free from albumin and sugar. She was placed upon a bread and water diet." (emphasis added)
"A week later, August 24, in spite of her rigid diet, she had gained 8 3/4 pounds more. (emphasis added) Her blood
pressure was 205/110 and she was having pains in the back of her head and was seeing specks before her eyes. The urine showed
a trace of albumin. No casts were found."(7)
Subsequently this poor woman had 11 convulsions. It is now clear that a "bread and water diet" is not effective prophylaxis
for MTLP!
In the very first volume of The American Journal of Obstetrics and Gynecology published 53 years ago [as of1974], Ehrenfest
reviewed "Recent Literature on Eclampsia," and found that venesection was still in common use for this dread disease: "For
the purpose of reducing the blood pressure and of eliminating toxins...."
He reported another then widespread approach: "Diuretics should be accompanied by a total or partial restriction of salt.
No meat shall be allowed." (emphasis added) Ehrenfest also noted the beginning of a scientific rejection of blood-letting
in the management of eclampsia: "Cragin says: Eclampsia patients after convulsions resemble so closely patients in shock,
that venesection seems illogical. They seem to need all the blood they have and more too."(8)
Here was the obvious clinical recognition of the hypovolemic shock which so commonly causes maternal and fetal deaths in severe
metabolic toxemia of late pregnancy.(9) The illogical use of salt diuretics in this disease may be viewed now as a "modern"
form of blood-letting in which electrolytes and water of the blood are forced out of the patient's body via her kidneys, a
kind of cell-free venesection!
In April, 1969, I presented a paper to an international meeting on "toxemia of pregnancy" in Basel, Switzerland, by invitation
of Dr. E.T. Ripperman, Secretary of the Organization Gestose.(10) Here I learned these interesting facts:
- Eclampsia has virtually disappeared from Switzerland; there had been no maternal death from this disease in Basel for
almost two decades.
- Some Swiss OB/GYN professors were still teaching that the pregnant woman must avoid red meat as prophylaxis against
eclampsia; for the Swiss this prescription seems to be working.
- The incidence of low birth weight babies born in the University Hospital, Basel, in the year 1967, from some 3,000 deliveries
was 3.0%.
It soon became apparent here from my discussions with many European OB/GYN authorities that the general nutritional status
of the peoples of Central Europe was exceptionally good, and that this adequate nutrition was the basic cause for the elimination
of severe MTLP and for the relatively low incidence of low birth weight infants.
My own paper presented in Basel was received with the utmost skepticism: the European obstetricians almost to the man responded:
"Surely there is no severe malnutrition in rich America." Surely? The incidence of low birth weight in our nation has risen
from 7.0% in 1950 to 10.0% now [1974] with much higher figures for all our poverty areas; MTLP continues to cause maternal-fetal
and newborn morbidity and mortality. Iatrogenic starvation during human pregnancy is still widely practiced throughout our
nation today because none of our medical or "public health" institutions have taken concrete actions to stop it.
A review of the unbound issues of The American Journal of Obstetrics and Gynecology reveals that for most of the 1950's and
1960's amphetamines and other "diet pills" were widely advertised for "weight control" in human pregnancy. Salt diuretics,
long recognized to be lethal to the severely toxemic patient and to her fetus, were promoted by this journal form 1958 to
1972. Professor Leon Chesley finally recognized their harmful effects on the maternal plasma volume.(11) The advertisements
for these water pills were then stopped but not their widespread use.
Today in 1973 the problems of rising prices for essential foods like lean meats, chicken, eggs, vegetables and fruits, and
the continuing poverty and economic depression in many areas of our nation can not be solved by the nation's physicians.
However, do not humane physicians today have a special and moral responsibility to stop the blind errors of iatrogenic
starvation in human pregnancy? Do not obstetricians, especially, in charge of human antenatal care in public clinics
and private offices, have a responsibility to their pregnant patients to give them scientific nutrition information? The
protective effects of applied, scientific nutrition in human antenatal care have recently been dramatically documented by
Mrs. Agnes Higgins of the Montreal Diet Dispensary.(12) The rationale for blind weight control to any "magic number" of pounds
in human pregnancy has been completely destroyed.(13) What then must the obstetricians of our nation do? What actions must
they take to insure maternal-fetal and newborn health for each woman who wants to produce a normal, full term child and remain
in good health herself?
- Recognize the complications of human pregnancy caused by malnutrition.(14)
- Teach each pregnant woman as a routine part of modern, scientific prenatal care, the basic principles of applied scientific
nutrition.
- Insure that she actually eats an adequate, balanced diet all through gestation.
- Encourage her to salt her food "to taste." (with rare exception)
- Stop "weight control" to any number of pounds. (when nutrition is adequate and balanced, the weight gain takes
care of itself with an average gain in healthy pregnancy of about 35 pounds)
-
Protect each pregnant woman and her unborn from all harmful drugs, especially salt diuretics and appetite depressants.
-
On the postpartum wards educate all pregnant patients who have suffered nutritional complications during pregnancy--so that
those complications will not recur in subsequent pregnancies.(15)
-
Stop iatrogenic starvation in human pregnancy.
These measures will begin to open a new era in preventive obstetrics in our nation and dramatically reduce the numbers of
low birth weight and brain-damaged and mentally retarded children now being born.
REFERENCES
- Hytten, F.E. and Leitch, I. The Physiology of Human Pregnancy. 2nd edition, Oxford, Blackwell Scientific Publications,
1970.
- Prochownick, L. "Ein Versuch zum Ersatz der Kunstlichen Fruhgeburt" (An attempt towards the replacement of induced premature
birth. Zbl. Gynak. 30:577, 1889.
- Williams, Sue Rodwell. Nutrition and Diet Therapy, 2nd Edition. St. Louis, Mosby, 1973, Chapter 17.
- Brewer, T.H. "Human Pregnancy Nutrition: an examination of traditional assumptions" Aust. N.Z. J. Obstet. Gynaecol. 10:87,
1970.
-
Pitkin, Roy M., Kaminetzky, Harold A., Newton, Michael, and Pritchard, Jack A. "Maternal nutrition: a selective review of
clinical topics" Obstet. Gynecol. 40:773-785, 1972.
-
Brewer, T.H. "Human maternal-fetal nutrition". Obstet. Gynecol. 40:868-870, 1972.
-
Rucker, M. Pierce. "The Behavior of the uterus in eclampsia" Amer. J. Obstet. Gynecol. 2:179-183, 1921.
-
Ehrenfest, Hugo. "Collective review: recent literature on eclampsia". Amer. J. Obstet. Gynecol. 1:214-218, 1920.
-
Brewer, T.H. "Limitations of diuretic therapy in the management of severe toxemia of pregnancy: the significance of hypoalbuminemia"
Amer. J. Obstet. Gynecol. 83:1352, 1962.
-
Brewer, T.H. "Metabolic toxemia of late pregnancy: a disease entity" Gynaecologia 167: 1-8, 1969. (Basel)
-
Chesley, Leon C. "Plasma and red cell volumes during pregnancy" Amer. J. Obstet. Gynecol. 112:440-450, 1972.
-
Primrose, T. and Higgins, A. "A study in human antepartum nutrition" J. Reproduct. Med. 7:257-264, 1971.
-
Pomerance, J. "Weight gain in pregnancy: how much is enough?" Clin. Pediat. 11:554-556, 1972.
-
Brewer, T. "Metabolic toxemia: the mysterious affliction." J. Applied Nutrition 24:56-63, 1972.
-
Brewer, T.H. "A case of recurrant abruptio placentae." Delaware Med. J. 41:325-331, 1969.
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